Rheumatoid Arthritis

Rheumatoid arthritis (RA) is the most common formreported. Beneficial results as far as signs and
of inflammatory arthritis affecting approximately 2.1symptoms were also demonstrated.
million Americans. It is a chronic, progressive,While this first study demonstrated both the safety
systemic, autoimmune disease for which there is noand efficacy of gene therapy, a more recent study
cure.confirmed it.
However, within the last 10-15 years, biologicDr. Christopher H. Evans and co-investigators
therapies aimed at the immune dysfunction thatreported their findings using a viral vector that carried
occurs in RA have enabled physicians to treatthe gene that blocks interleukin-1, another protein
patients with much more success… in fact,that promotes inflammation and causes cartilage
often allowing the achievement of remission.breakdown, in the February issue of Human Gene
Gene therapy approaches have been employedTherapy.
recently in the hopes that patients who do not”Arthritis is a good target for (gene therapy)
achieve remission may still yet get needed relief.because the joint is a closed space into which we
The first gene therapy approach was developed bycan inject genes,” was a statement issued by
Targeted Genetics in Seattle, Washington. TheirEvans.
compound, tgAAC94 was developed as a potentialUnlike the tgAAC94 study, tissue was removed from
supplement to systemic anti-TNF-alpha (anti-tumorthe knuckle joints of two patients with severe RA
necrosis factors drugs such as Enbrel, Humira, andand a harmless virus was inserted into the tissue
Remicade have become the standard of care forcells, in order to serve as a vector to carry a gene
rheumatoid arthritis) therapy for use in patients withthat blocks action of the interleukin-1 protein to the
inflammatory arthritis who had one or more jointsjoint. After being placed in culture to grow and
that did not respond to systemic therapy.multiply, the cells were injected back into the afflicted
The product used a recombinant adeno-associatedjoints.
virus to deliver a DNA sequence that coded for aOne patient who received gene therapy in two joints
form of the TNF-alpha receptor (TNFR). TNFR blocksexperienced an 85 percent reduction in pain in one
the immune stimulating activity of TNF-alpha. Directjoint within 1 day, and both joints were pain-free
injection of tgAAC94 into affected joints leads tofrom 1 week onward. Remarkably, the researchers
the localized production of soluble TNFR within jointreport, joints receiving the therapy were protected
cells, reducing the activity of TNF-alpha within thefrom flares that occurred during the study period.
joint and, potentially, leading to a decrease in theThe second patient also responded to gene therapy,
signs and symptoms of disease and stopping jointwith a 70 percent reduction in pain between weeks 2
destruction.and 3.
The adeno-associated virus is a naturally occurringThis paper, therefore, also showed that painful
virus that has not been associated with any diseasesymptoms can be lessened through gene therapy.
in humans.Both of these studies have demonstrated that gene
Data from an initial Phase I trial demonstrated thattherapy is feasible in humans. More importantly, it has
injecting tgAAC94 into joints was safe andbeen shown that this approach may be helpful for
well-tolerated in patients taking conventional diseasedelivering biologic agents into stubborn joints that do
modifying anti-rheumatic drugs (DMARDS).not respond to systemic therapies.
No drug-related serious adverse events were