Ubiquinol - The Other Half of the CoQ10 Story

Ubiquinol, the reduced form of Coenzyme Q10No discussion of CoQ10 would be complete without a
(CoQ10), is a recent addition to the supplementbrief mention of its documented health benefits.
offerings of several companies, and one which hasSupplemental CoQ10 (especially at therapeutic doses)
generated as much confusion as it has excitement.has been the subject of numerous studies over the
As a supplement, ubiquinol is fairly new. As a criticallast half century, particularly in applications for
part of human metabolism, our knowledge ofcardiovascular health. Several studies have shown
ubiquinol dates back nearly as far as the discovery ofbenefits of CoQ10 in patients diagnosed with chronic
CoQ10. While CoQ10 is often thought of as a "static"heart failure, exercise-induced angina, hypertension, or
nutrient within the context of nutrition, it is actuallythose who have recently experienced infarction.
one which dynamically interconverts between twoThere is also early evidence that CoQ10 may protect
useful states (the oxidized "ubiquinone", and thethe heart from damage during chemotherapy, bypass
reduced "ubiquinol"). In this article, we will discusssurgery, or in diabetes. Aside from cardiovascular
ubiquinol as part of the CoQ10 "cycle", and how thisapplications, CoQ10 has also been studied for its
cycle has important consequences in humanpotential benefits for other conditions which involve
metabolism. Additionally, we will discuss how ubiquinoldysfunctions in cellular energetics , neurological
as a supplement may take advantage of this cycledegeneration, or oxidative damage (including
to exhibit increased assimilation into the body.Parkinson's and Huntington's disease, ALS and
What is CoQ10?periodontal disease).
Coenzyme Q10 or CoQ10 is a member of a family ofWhile the clinical evidence for the potential benefit of
important biological compounds called ubiquinones. It isCoQ10 in many of these applications shows promise,
a lipophilic, water-insoluble substance whichvariability in study outcomes necessitates further
participates in a variety of biochemical oxidation andresearch in these areas before definitive
reduction reactions (redox reactions). First identified inrecommendations can be made. Results of more
1957 as an essential component of the energyrecent studies have revealed that CoQ10 dosages
production system in cells, CoQ10 and other(particularly in terms of circulating plasma levels) may
members of the ubiquinone family have since beenhave been too low in earlier trials to demonstrate
identified as critical metabolic compounds in a rangepositive clinical benefits. Particularly in terms of
of aerobic organisms. Indeed, ubiquinones arecardiovascular health, CoQ10 blood levels of greater
ubiquitous.than 2.5 mcg/ml appear to have more consistent
Because of its critical role in metabolism, humansresults in improving cardiovascular function. To put
have the ability to synthesize their own CoQ10 (eg. itthis in perspective, a recent study of CoQ10
is nutritionally non-essential), although modestsupplementation in heart failure patients required a
amounts can be obtained through diet or asdosage of 300 mg of ubiquinone per day (in a
supplements. In humans, CoQ10 is found in each cellhigh-absorption delivery system) for one month to
in the body, but is particularly concentrated in tissuesexceed this threshold and exhibit improvements in
which have large energy requirements (like the heart,cardiovascular parameters. Future clinical studies of
liver, kidneys, and skeletal muscles), with smallerCoQ10, designed with this threshold in mind, should
amounts in the brain, lungs, and intestines. There arecontinue to add to the growing body of evidence of
also substantial amounts of CoQ10 in circulation, mostCoQ10's positive role in preserving proper
often associated with lipoprotein (LDL or HDL)cardiovascular function. And as we shall see in the
particles. The total CoQ10 pool in a normal adult hasnext section, the development of supplemental
been estimated to be between 0.5 and 1.5 grams.ubiquinol may facilitate our ability to deliver CoQ10 at
Within cells, about half of the cellular CoQ10 is foundtherapeutically-relevent levels.
within the mitochondria (the energy-producing centersUbiquinol as a Supplement: Additional Benefits?
of cells), and this is where the final steps of CoQ10As we have seen previously, CoQ10 functions by
synthesis occur. Extramitochondrial CoQ10 (that is,cycling between two stable forms, ubiquinol and
CoQ10 which is located in areas of the cell which areubiquinone. The outcomes of this cycle are the
NOT charged with producing cellular energy) cangeneration of cellular energy, and the protection of
amount to 50-60% of the total CoQ10 pool. CoQ10 ismembranes and lipids from oxidation. Dietary or
found throughout cell membranes, as well as in othersupplemental CoQ10 also participates in this cycle.
cellular structures (organelles) such as nucleus,Dietary ubiquinone (oxidized CoQ10; the form most
cytoplasm, and endoplasmic reticulum.commonly found in foodstuffs and until recently,
Experimentation has also revealed that while the finalsupplements) can be reduced to ubiquinol during its
steps of CoQ10 production occur in the mitochondria,transit from the gut to the bloodstream.
it can be exported to other subcellular locations.If CoQ10, regardless of form or source, is still able to
Similar studies have shown that exogenous (obtainedparticipate in the redox cycle, then what benefit
from the diet) CoQ10 incorporates not only into thewould supplemental ubiquinol possibly have? The
mitochondria, but into the plasma (outer) membraneanswer to this question requires an understanding of
of cells as well as other intracellular membranes.CoQ10 pharmacokinetics (the particulars of CoQ10's
One Role, Two Forms, Many Consequencesabsorption, utilization, and elimination). As it turns out,
In the course of participating in various oxidation anddietary or supplemental ubiquinol may have a distinct
reduction reactions, CoQ10 itself is cycled betweenadvantage over ubiquinone in its facility of absorption.
two stable states (called a "redox pair"): a fullyLike many fats and lipophilic nutrients, CoQ10
oxidized form ("ubiquinone"), and a fully reduced form(regardless of form) is usually taken up by the
(called "ubiquinol"). CoQ10 cycles through theseintestinal enterocytes (cells which line the intestines),
oxidated/reduced forms in order to achieve itspackaged into lipid particles, and released into the
metabolic goals. The CoQ10 cycle is simple: Ubiquinonelymphatic system. From the lymphatic system, these
picks up electrons and becomes ubiquinol. Ubiquinolparticles are shuttled into circulation, where they are
release its electrons and becomes ubiquinone again.free to be transported throughout the body to
And the cycle repeats.where they may be needed. The absorption of
It would appear, therefore, that CoQ10 has a verydietary CoQ10 is quite poor. It has limited solubility in
simple function - to move electrons. Why is thelipids, and its highly variable absorption depends on
movement of electrons important? This seeminglythe other contents of the gut (for example,
trivial feat has important consequences in metabolism;absorption is lower on an empty stomach and
the transfer of electrons is a fundamental step in thegreater when taken with food of high lipid content).
production of energy, in the regeneration ofSome studies have measured absorption as low as
antioxidants in cell membranes, and in the2-3% of the total dosage.
construction of other important biological molecules.One of the most exciting consequences of the
Each cell in the body needs a source of energy todevelopment of a stabilized dosage form of ubiquinol
survive, so cells break down sugars, fats, and aminois its innate ability to be absorbed more efficiently
acids to make energy. In the mitochondria of ourthan ubiquinone. There is experimental evidence that
cells, CoQ10 is abundant, carrying electrons to aid inCoQ10 must be reduced (to ubiquinol) in intestinal
the chemical reactions that "burn" (oxidize) cellular fuelenterocytes prior to release into the lymphatic
to produce chemical energy in the form of adenosinesystem; this coupled absorption/reduction may be a
triphosphate (ATP). Since adequate amounts of ATPrate limiting step in CoQ10 assimilation. Dietary
are essential for powering our cellular machinery, it isubiquinol (which is already reduced) avoids this
easy to understand the importance of CoQ10 inreduction reaction, and may be directly available for
human metabolism. Both forms of CoQ10 areabsorption. This may explain why ubiquinol-based
necessary to shuttle electrons betweenCoQ10 supplements exhibit enhanced bioavailability
energy-producing reactions (ubiquinone picks them up,over ubiquinone supplements. Preliminary testing in
and ubiquinol drops them off).humans indicates at least double the absorption of
Outside of the mitochondria, CoQ10 performs aubiquinol over ubiquinone. Comparisions of blood levels
slightly different role as a membrane andbetween trials, while less accurate, estimates the
lipid-associated antioxidant. About half of the humanimprovement in absorption to be significantly higher.
body's total CoQ10 pool may be functioning in thisFuture clinical studies (designed specifically to
capacity. CoQ10 is one of the major antioxidantcompare the absorption of these forms) are required
constituents of the LDL particle. In its reduced formto more accurately quantify ubiquinol's enhanced
(ubiquinol), CoQ10 can be found alongside alpha- andabsorption, and what effect patient age or medical
gamma-tocopherol, lycopene, and beta-carotene incondition may have on this value.
LDL particles as resident antioxidants to prevent LDLSummary
oxidation. Coenzyme Q10 is one of the firstCoQ10, a critical biological molecule in the metabolism
antioxidants to be depleted when LDL is subjected toof aerobic organisms, owes its utility to its ability to
oxidation. Studies have implicated ubiquinol as acycle between two relevent forms, ubiquinone and
regenerator of oxidized tocopherol within the LDLubiquinol. This cycle takes a central role in several
particle; studies of hypercholesterolemic elderlymetabolic reactions, notably the production of cellular
patients have equated highly oxidized LDL particlesenergy and the protection of membranes and lipids
with low ubiquinol levels. In its role as an antioxidant,from oxidative damage. Sufficient levels of CoQ10
the reduced form of CoQ10 (ubiquinol) functions asare an important factor in human metabolism
the active antioxidant, giving up its electrons to other(particularly in the maintenance of cardiovascular
oxidized molecules (like oxidized tocopherol) tohealth) and clinical research in this area continues to
regenerate them. This process converts ubiquinol tostrengthen this observation. Ubiquinol (the reduced
ubiquinone. The newly formed ubiquinone must thenform of CoQ10) as a supplement or therapeutic may
be converted back into ubiquinol in order to becomebe an advantageous development. Its advantage
reengaged in the antioxidation process, although themay not have a direct effect on the well-regulated
mechanism by which this occurs within LDL particles isCoQ10 redox cycle in healthy people, but rather its
not yet known.ability to circumvent one of the possible limitations of
Health Benefits of CoQ10CoQ10 absorption.